|
Back to Ranvet 500
Plus Plus
For late Pregnant and Lactating
Mares
Ranvet 500 Plus provides pregnant mares with the building blocks for sound
foetal bone and tissue development to provide a solid foundation for
early maturity. Ranvet 500 Plus raises the initial quality of mares' milk
and extends the length of lactation by balancing the total mineral and
protein intake.
Foals, Weanlings and Yearlings
Many of the bone problems and much of the unsoundness that plague
breeders and trainers - the sprains and splints - are the tragic
result of improper skeletal development during the foetal and early
growth stages.
A daily measure of Ranvet 500 Plus will
provide the balancing nutrients for sound growth, but the challenge
for the stud manager is to CONTROL the amount of grain in the feed.
For the total feed intake to meet the requirements for growth it is
fundamental to increase the amount of grain fed in colder months and
decrease the grain component when grasses put up a head of seed. But
the daily intake of mineral, amino acid and vitamin supplement should
not decrease.
How to use Ranvet 500 Plus
Ranvet 500 Plus is a complete protein, amino acid, mineral and vitamin
supplement combined into a single pellet. You add your own grain and
chaff. Simply combine Ranvet 500 Plus with the amount of grain you need to
feed; either mix your own oats plus chaff and add a measure of Ranvet
500 Plus or simply add Ranvet 500 Plus to your own grain mix.
Early introduction of a creep feed
actually induces self-weaning. Start foals with 50-100g of Ranvet 500
Plus
mixed with steam rolled oats, salt and chaff plus 15ml or Ranvet IRON
PLUS. The sweet iron syrup will attract foals to the creep feeder.
Nutritional Information
Protein 31.2%, Oils 3.7%, Fibre 8.8%, Ash 22.8%, Calcium 5.75%,
Phosphorus 1.26%, Copper 210mg/kg, Zinc 350mg/kg, Vitamin A 25,000
IU/kg, Vitamin D 17,000 IU/kg.
Ranvet 500 Plus is a complete protein, amino
acid, mineral and vitamin supplement combined into a single pellet.
It adds a balanced spectrum of amino acids to deliver full value
from the protein in grasses and legumes and at the same time
balances the minerals in the feed to lay down dense bone. With
Ranvet 500 Plus the amount of grains in the diet can be increased or
decreased to control the growth rate of growing foals.
The aim of Ranvet 500 Plus is to achieve optimum size and development at
all stages of the young horse’s growth, with the ultimate goal being
to produce a more fully developed and stronger yearling capable of
performing as an early two year old. To achieve this aim, the rapid
growth of the foetus must be catered for during the last quarter of
gestation, the quality of the mare’s milk must be high and
maintained and the nutritional needs of the rapidly growing foals
and weanlings must be catered for.
Developmental orthopaedic disease (DOD) is a term used to describe a
range of skeletal problems associated with growth and development of
the foal including osteochondritis dessicans (OCD), physitis,
angular limb deformities, flexural deformities (contracted tendons,
club foot), subchondral cystic lesions (bone cysts), cervical
vertebral malformation (wobbler syndrome) and cuboidal bone
malformation. While not all cases of these disorders will be a
problem of development, most authorities agree that developmental
problems lie behind the cause of the majority of cases. It has been
estimated that DOD collectively costs the Australian Thoroughbred
industry $60 million each year1. A correlation between the
occurrence of DOD in horses and reduced amounts of calcium,
phosphorus, zinc and copper, but not with the amount of other
nutrients in weanling’s diets has been observed2.
Nutritional imbalances as a cause of DODs include excess dietary
energy, imbalanced dietary protein, calcium and phosphorus
imbalances and inadequate copper and zinc.
• Excess energy intake, particularly in the form of soluble
carbohydrates will consistently produce lesions of OCD which can
occur within a few months3. Overfeeding the pregnant mare may also
affect the incidence of DOD in her foals1.
• While DODs occur most commonly in the overfed horse, it may also
occur in the underfed horse. This is thought to occur as a result of
inadequate protein in the diet, resulting in impaired bone and
muscle development in the horse1,3.
• Calcium and phosphorus together make up approximately 70% of the
mineral content of the body and 50% of the skeleton. A deficiency of
calcium or phosphorus will cause impaired endochondral ossification
and decreased bone mineralisation, resulting in a wide range of
skeletal disorders. The diet must not only contain adequate amounts
of calcium and phosphorus, the animal must be able to absorb and
utilise these nutrients. A sufficient excess of either mineral will
decrease the absorption of the other. Ideally, the ratio of calcium
to phosphorus should be between 1.2:1 and 2:1 however young horses
will tolerate between 0.8:1 to 3:1. Outside this range, alterations
in endochondral ossification, resulting in DOD may occur.
• Copper is involved in stabilising bone collagen and elastin
synthesis. A copper deficiency impairs these functions, resulting in
developmental orthopaedic disease (DOD)3. A correlation between the
occurrence of DOD in horses and reduced amounts of calcium,
phosphorus, zinc and copper, but not with the amount of other
nutrients in weanling’s diets has been observed2. The incidence of
these diseases decreased significantly when these minerals,
particularly copper, were increased in the diet. Copper
supplementation of young foals has been shown to reduce the
prevalence of cartilage lesions4,5. Furthermore, supplementation of
pregnant mares decreased radiographic indices of physitis in the
distal third metatarsal bone of foals and the prevalence of
articular cartilage lesions6. Dietary zinc deficiency has also been
postulated as a cause of DOD2. The National Research Council
publication “Nutrient Requirements of Horses” (1989), recommends
that all horses receive 10 ppm (parts per million) copper and 40ppm
zinc.
However, research indicates that growing horses consuming these
levels (or lower) are more likely to develop DOD. Despite other
studies suggesting that lower amounts of dietary copper and zinc may
be adequate, because of studies suggesting that increasing copper
and zinc in the growing horse’s diet may decrease the risk and
occurrence of DOD, and because there is no risk of harm from doing
so, it has been recommended that the levels of copper and zinc added
to the growing horse’s diet be increased to between 25 and 40ppm
copper and between 40 and 60ppm zinc.
Ranvet 500 Plus has been developed through a series of research trials
and has been specifically formulated for the growing foal and
pregnant mare. The product is being continually updated as new
research findings on DODs emerge. The use of Ranvet-500 has resulted
in excellent growth rates as shown below.
* Source of data: Journal of Animal Science (1995) 75, 2513-2517
Ingredients: Oilseed products
and by-products, Minerals, L-Lysine, Oils and Fats, Dried forages,
Dextrose and Molasses, DL-Methionine, Milk products, Vitamins,
permitted antioxidants, Flavouring and non-prohibited substances to
improve nutrient utilization.
| Recommended
Daily Feeding Rates |
|
| |
|
| Mares
in late pregnancy/lactation |
650g |
| *Plus
30mL Ranvet IRON PLUS |
|
| |
|
| Creep
fed foals* |
|
| At
2-4 weeks |
50-100g |
| At
two months |
150g |
| At
four months |
250g |
| *Plus
30ml Ranvet IRON PLUS |
|
| |
|
| Weanlings |
|
| At
six months |
350g |
| At
nine months |
500g |
| |
|
| Yearlings |
650g |
Pack
Sizes
Ranvet 500 Plus is available in a 20kg bag which includes a
convenient 300g measuring cup |
Group
One Seal of Quality
Ranvet 500 Plus puts your horses on the cutting edge of high
performance feeding. Products that carry the Group One Seal of
Quality are formulated to meet the requirements of racing at
Group One level - the highest level of competition - by Ranvet;
the name you can trust for quality. |
References
1Aldred, J., 1998. Developmental Orthopaedic Disease in Horses.
RIRDC Publication No. 97/79
2 Knight, D.A., Gabel, A.A., Reed, S.M. et al (1985). Correlation of
dietary mineral to incidence and severity of metabolic bone disease
in Ohio and Kentucky. Am. Assoc. Pract. Proc, 445-461.
3 Lewis, L.D., 1995. Developmental Orthopaedic Diseases in Horses.
In: Equine Clinical Nutrition. Eds: Cann, C., Hunsberger, S.,
Williams and Wilkins, USA
4 Knight, D.A., Weisbrode, S.E., Schmall, L.M., Reed, S.M., Gabel,
A.A., Bramlage, L.R., Tyznik, W.I. (1990). The effects of copper
supplementation on the prevalence of cartilage lesions in foals.
Equine Vet J. 22, 426-432.
5 Hurtig, M.B., Green, S.L., Dobson, H. et al (1990). Defective bone
and cartilage in foals fed a low copper diet. Am. Assoc. Equine
Pract. Proc., pp 637-644.
6 Pearce, S.G., Firth, E.C., Grace, N.D., Fennessy, P.F. (1998c).
Effect of copper supplementation on the evidence of developmental
orthopaedic disease in pasture-fed New Zealand Thoroughbreds. Equine
Vet J. 30, 183-185.
NRC (1989). Nutrient Requirements of Horses, 5th Edn. National
Academy Press, Washington, D.C. |
|
|
|